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Abstract

Although a single session of low to moderate intensity exercise induces post-exercise hypotension (PEH), the effect of blood flow restriction (BFR) exercise on PEH is still unknown. PURPOSE: We tested hypothesis that aerobic exercise with BFR induced (PEH), and the reduction in blood pressure (BP) was due to peripheral vasodilation via the histamine receptors. METHODS: Ten male subjects participated in this study. The subjects were randomly assigned to walk on the treadmill for 10 minutes at 6.4 k/m, 0% grade with or without BFR after taking histamine receptor blockade. Following exercise, BP was measured at 10 min interval for 60 minutes. Heart rate (HR), stroke volume (SV), cardiac output (CO), mean arterial pressure (MAP), and total peripheral resistance (TPR) were evaluated. RESULTS: Our results indicated that MAP was significantly lowered immediately after exercise at 20 minutes, 30 minutes, and 40 minutes before the blockade compared to after the blockade. The significant reduction in diastolic blood pressure (DBP) occurred. There were no significant differences in HR, SV, CO, and TPR between before the blockade and after the blockade. MAP was substantially decreased at 20 min, 30 min, and 40 min before the blockade compared to the resting (-4±1, -3±1, and -4±1, respectively) while increasing MAP after the blockade. CONCLUSION: The current study demonstrated that low-intensity aerobic exercise with BFR lowered MAP via histamine receptor-induced peripheral vasodilation. The combined histamine H1 and H2 receptors play a pivotal role in sustained postexercise hypotension following BFR exercise.

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